Chiara Beretta
Doktorand vid Institutionen för folkhälso- och vårdvetenskap; Molekylär geriatrik/ Rudbecklaboratoriet
- Telefon:
- 018-471 34 92
- Mobiltelefon:
- 073-469 75 04
- E-post:
- chiara.beretta@pubcare.uu.se
- Besöksadress:
- Dag Hammarskjölds väg 20
- Postadress:
- Rudbecklaboratoriet
751 85 Uppsala
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Publikationer
Urval av publikationer
- Long-term effects of amyloid-beta deposits in human iPSC-derived astrocytes (2023)
- Intracellular deposits of amyloid-beta influence the ability of human iPSC-derived astrocytes to support neuronal function (2023)
- Amyloid-beta accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism (2023)
- Enhanced neprilysin-mediated degradation of hippocampal A beta 42 with a somatostatin peptide that enters the brain (2021)
- Traumatic brain injury in the presence of Aβ pathology affects neuronal survival, glial activation and autophagy (2021)
- Extracellular vesicles from amyloid-beta exposed cell cultures induce severe dysfunction in cortical neurons (2020)
- Long-term effects of amyloid-beta accumulation in human iPSC-derived astrocytes
- Amyloid-beta accumulation in astrocytes affects their impact on neuronal function in a human iPSC-based model of Alzheimer’s disease
Senaste publikationer
- Amyloid-β deposits in human astrocytes contain truncated and highly resistant proteoforms (2024)
- Long-term effects of amyloid-beta deposits in human iPSC-derived astrocytes (2023)
- Intracellular deposits of amyloid-beta influence the ability of human iPSC-derived astrocytes to support neuronal function (2023)
- Amyloid-beta accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism (2023)
- Enhanced neprilysin-mediated degradation of hippocampal A beta 42 with a somatostatin peptide that enters the brain (2021)
Alla publikationer
Artiklar
- Amyloid-β deposits in human astrocytes contain truncated and highly resistant proteoforms (2024)
- Long-term effects of amyloid-beta deposits in human iPSC-derived astrocytes (2023)
- Intracellular deposits of amyloid-beta influence the ability of human iPSC-derived astrocytes to support neuronal function (2023)
- Amyloid-beta accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism (2023)
- Enhanced neprilysin-mediated degradation of hippocampal A beta 42 with a somatostatin peptide that enters the brain (2021)
- Traumatic brain injury in the presence of Aβ pathology affects neuronal survival, glial activation and autophagy (2021)
- Extracellular vesicles from amyloid-beta exposed cell cultures induce severe dysfunction in cortical neurons (2020)
- Astrocytes with Alzheimer’s disease pathology provoke lipid droplet mediated cell-to-cell propagation of MHC II complexes
- Long-term effects of amyloid-beta accumulation in human iPSC-derived astrocytes
- Amyloid-beta accumulation in astrocytes affects their impact on neuronal function in a human iPSC-based model of Alzheimer’s disease
